MEDICAL - PATHOLOGY

• Document Type: CREST
• Collection: General CIA Records
• Document Number (FOIA) /ESDN (CREST): CIA-RDP80-00809A000600221008-8
• Release Decision: RIPPUB
• Original Classification: S
• Document Page Count: 3
• Document Creation Date: December 22, 2016
• Document Release Date: June 28, 2011
• Sequence Number: 1008
• Publication Date: May 12, 1949
• Content Type: REPORT

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CLASSIFICATION SE(AU
CENT . INTELLIGENCE AGENCY
INFORMATION FROM
FOREIGN DOCUMENTS OR RADIO BROADCASTS CD NO.
COUNTRY Russia
SUBJECT Ne,dical ? Pathology
HOW
PUBLISHED Weekly newspaper
WHERE
PUBLISHED Moscow
DATE
PUBLISHED 13 Apr 1949
LANGUAGE Russian
DATE OF
INFORMATION 1949
DATE DIST. 42-May 1949
NO. OF PAGES 3
SUPPLEMENT TO
REPORT NO.
THIS IS UNEVALUATED INFORMATION
Prof D. Roseiyskiy
Honored Worker of Science
Pneumonia is a diwease which most frequently oamplicates in."luenzal
oases. The influenzal viruses cause hyperemia of the bronchial passages
and of the pulmonary parenchyma accompanied by traumatic conditions of the
vascular walls and by hemorrhage. Such conditions increase the vital
activity of various microbes, such as pnemmocMOus, Pfeiffer'e bacillus,
streptococcus, etc., and increase the possibility of contracting pneumonia.
Pneumonia of such etiology frequently develops into the hemorrhagic type
and Is characterized by hemoptysts.
Preliminary examinations during the Initial stage disclose no
pulmonary deganerations. It is possible, however, to ausculate weak
crepitatione in the lungs. With further development of pneumonia, the
patient tussiouilatee and develops definite dyspot.ea. Expectoration per-
sista, leading to pyoptysis and, frequently, to hemoptysis.
It must be noted, however, that in many cases the exact source of the
crepitation is impossible to determine. Many specialists failed to deter-
mine the source even with the- aid of bron:hcphonss on the basis of bronchial
respiration.
It is possible to determine the nidus with the gr9dual progress in the
individual case. Inf!uenzal pneumonia diffuses veiy rapidly in the lung
and adny cases, having only a few nidi in the morning, develop a general
infection of the lunge by the same evaair3.
The acute stages of pneumonia frequently coincide with the onset of
influenzal .omplication. This co:idition is usually seen following 'luv
initial drop in temperature, or 4 or 5 days after the onsei, of the ,i.+sease.
The clinical picture of influenzal pneumonia cases is very eizzilar to that
of cases afflicted with acute pnemoris.
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The course oY the d
iseaseis usually slow and takes ? or 4, if not
more, weeks before any improvement can be seen and before the tempera-
ture commences to drop. However, in weakened cases and in aged persons,
pneumonia is liable to linger for several months. In such cases there is
the additional danger of tubercular development. The progress of pneu-
monia in aged persons is sometimes unaccompanied by the usual symptoms;
many with the disease have no tussis or abnormal expectoration and have
subnormal or even normal temperature. Physical examinations result in
the detection of scattered sources of muffled sound, bronchial respiration,
and crepitation.
In recent years, several atypical forms of influenzal pneumonia have
been encountered. These include those having virus etiology, such as
peripheral or central pneumonia, with a characteristic morphological and
clinical picture. The microorganisms invade the interior of the lobes or
are localized on the p^riphery of the lungs. These types of pneumonal
afflictions are serious and are frequantly the initial stage and not the
secondary stage, of influenza. Physical symptoms are insignificant and the
few who are examined cannot be diagiosed by roentgenological exsminatior-e.
In the majority of the cae3e there is a light crepitation.
Patients afflicted with peripheral and central nneumnnIa.be nme
tuasicular and frequently develop hemoptysis. They also complain of
cephalalgia and fever. In the intial stage of this disease the tussle
are espentorated. Microscopic eXamination shove the presence of Aiplococcus,
frequently very d-'ffionlt'to aet',rmine and the only metaou of diagnosis of
graphic picture. For example, vnen a patient indicates cropitation and
bronoh3.al respiration, roentgenograms show no histologic changes in the
lungs. Frequently, when there is no clinical symptom and an absence of
intoxication, the roentgenograms remain unchanged for a long period of time.
All that can be recommended is that cases whose roentgenograms show persist-
ent shadows should be confined to bed until the infective nidue subaides.
Severe cases of influental infection of z septic character may produce
specific roentgenograms somewhat similar to disseminated tuberculosic or
miliary caroinoamtosis. Actually, therefore, roentgenogram eerre only to
locate the site of pnetuoonal infection. which is too deeply seated to be
determined by 1.ercussible or aurculatory methods.
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Iniluenzal pneumonia can be complicated by interlobular infection,
pulmonary abscesses, and, in some cases, even pulmonary gangrene. Long
series of studies shoved that in pneumonal cases complicated with in-
f iuenzal infection, it is not uncommon to find abscessed conditions.
Ausculatatory methods are ineffective in the diagnosis of small 'aultiple
pulmonary abscesses.
When an influenza) pneumonia case succumbs in one to 3 days after the
onset of the disease, it is safe to assume that pneumonia has been present
for several months.
pneumonia case, registering no febrile temperature. The infective process
generally commences in the lower regions of the lunge and is strictly local-
ized. Infrequently there is an increased crepitation caused by the friction
of the pleura. However, an analysis of the blood indicates a high white
count and an increase in the polymorphonuclear neutrophilic leukocyte count.
More often than not, influenzal pneumonia is complicated by pleurisy
which mr y be bilateral, dry, fibrinous, serous, suppura'ira, or hemorrhagic.
Snifemide m.eperatIo a were edminietarev to cases with irftetenv!al
pneumonia in 1943, 1944, and 1946. These primary sulfemide compounds were
suifathiazol and sulfadiazine which were gi'en together with urotropic
compounds. However, in the past year there vere many who reacted nega-
tively to sulfamide compounds and many cases indicated no favor:-ble re-
action to penicillin administrations.
Due to the morbid action of thr, virus of influenza on the cardiovascular
system, It is vital that We systfsa be stimulated during the early stage of
the disease. Administration of such substances as camphor or caffeine is
recommended.
Immediately following an irritation of the respiratory tracts which
causes tussle, it is necessary to administer such narcotics as codeine,
dionin, Dover's powder, as well as hot milk with alkaline admixtures. For
vet crepitation, the administration of ipecacuanha, eenege, thera?psie, or
marshmallow root Is recommended. Prompt medication is recommended because
harsh expectoration may lead to hemoptyais due to the hematologic condition.
It ilea been noticed, particularly in cases indicating fever, that the
temnereture decreases after the cessation of tussle and =cold and prrulant
expectoration. Favorable rebulte in lowering the temperature have been ob-
tained with heavy dosages of streptoolde, 0.3 grams, up to six times a day.
Subcutaneous administration of camphor produced favorable results in in-
fluenzal cases complicated with capillary bronchitis or pneumonia.
Examinations shoved. that with the formation of lesions, as much as
300 cc of 1,10*' may flood the lungs and cut the oxyger. intake in the lung
tissue.
In Tier of the severity of this diaerse, which lovers the to :us of
the cardiovascular system, it is recommended that patients, after their
recovery, remain for some time under the observation and care of a physician.
One more word of warning. Influenza) pneumonia, even in a very mild
form, is highly contagious and acts as the basis for possible development of
any one of several serious and possibly fatal diseases.
50X1-HUM
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